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Hiroshi YAMAUCHI, Researcher | Cited by 5,018 | of Kyoto University, Kyoto (Kyodai) | Read 148 publications | Contact Hiroshi YAMAUCHI.
Studies using animal experiments have shown secondary neuronal degeneration in the thalamus after cerebral infarction. Neuroimaging studies in humans have revealed changes in imaging parameters in the thalamus, remote to the infarction. However, few studies have directly demonstrated neuronal changes in the thalamus in vivo.
- 10.1161/STROKEAHA.122.038846
- 2022/10
- Stroke. 2022 Oct; 53(10): 3153-3163.
Mar 22, 2007 · Citing Literature. Volume 61, Issue 5. May 2007. Pages 454-465. <em>Annals of Neurology</em> is a leading neurology journal from the American Neurological Association focusing on diseases of the human nervous system.
Correspondence to: Hiroshi Yamauchi, MD, PhD, Department of Psychiatry, Graduate School of Medicine, Kyoto University, 54 Shogoin-Kawahara-cho, Kyoto 606- 8507, Japan. Email yamauchi@kuhp.kyoto-u.ac.jp
Jun 2, 2015 · Abstract. In patients with atherosclerotic internal carotid artery or middle cerebral artery occlusive disease, chronic reduction in cerebral perfusion pressure (chronic hemodynamic compromise) increases the risk of ischemic stroke and can be detected by directly measuring hemodynamic parameters.
Jan 1, 2014 · Hiroshi Yamauchi MD, PhD 6 Abstract In patients with atherosclerotic internal carotid artery (ICA) or middle cerebral artery (MCA) occlusive disease, fluctuation of cerebral perfusion in the condition of chronic hemodynamic compromise may cause not only cerebral infarction but also selective neuronal damage in the cerebral cortex that is not ...
Hiroshi Yamauchi, MD; Hidenao Fukuyama, MD; and Jun Kimura, MD. Background and Purpose: The pathophysiology of crossed cerebellar diaschisis remains to be elucidated. The mechanism responsible appears to be deafferentation through the corticopontocerebellar tract, which terminates in the cerebellar gray matter.
