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      • More education did not protect individuals from developing neurodegenerative and vascular neuropathology by the time they died but it did appear to mitigate the impact of pathology on the clinical expression of dementia before death.
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  2. Jul 23, 2010 · Examining the brains of 872 people who had been part of three large ageing studies, and who before their deaths had completed questionnaires about their education, the researchers found that more education makes people better able to cope with changes in the brain associated with dementia.

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  3. How can a relatively small number of years of formal education occurring early in life affect risk for dementia in old age? This review advances the literature by providing a broad systematic review of both dementia prevalence and incidence studies.

  4. However, this has not been well studied. Although some studies reported association between lower educational attainment and higher dementia, these alleged effects of education may not be specific for dementia but represent an alternative pathway from negative lifestyle to increased incidence of age-related cognitive impairments [14–16].

  5. Yet, much remains unknown as to how early life factors, particularly early life cognitive function, and early life environments, such as family environments, help explain why education so robustly protects against later life dementia.

  6. Jul 30, 2020 · Differences in early-life education linked to dementia risk. Research presented at the 2020 Alzheimer’s Association International Conference suggests that higher quality early-life education is linked to better language and memory performance, and lower risk of dementia.

  7. Dec 1, 2020 · This systematic review has updated and reiterated the evidence for an association between education in early life and reduced risk of AD and any dementia incidence, with the addition of 16 previously unconsidered studies.

  8. Early-life (younger than 45 years) risks, such as less education, affect cognitive reserve; midlife (45–65 years), and later-life (older than 65 years) risk factors influence reserve and triggering of neuropathological developments.

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