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  1. Evidence of dysrhythmias reported in this systematic review reflect how excessive alcohol consumption plays a destructive role on the myocardial cells, consistent with dilation of the heart chambers, a known consequence of alcohol toxicity in both acute and chronic excessive alcohol consumption (Voskoboinik, Prabhu, Ling, Kalman, & Kistler, 2016). For example, alterations in electrolyte ...

  2. May 20, 2022 · Alcohol is often cited to be a common cause of cardiomyopathy and heart failure. However, in most available population-based studies, a modest-to-moderate alcohol consumption has been associated with favorable effects on the cardiovascular system, including a lowered risk of heart failure, compared with no alcohol consumption. Available genetic epidemiological data have not supported a causal ...

  3. Aug 8, 2023 · Alcoholic cardiomyopathy is a leading cause of non-ischemic dilated cardiomyopathy in United States. Incidence of alcoholic cardiomyopathy ranges from 1-2% of all heavy alcohol users. It is estimated, approximately 21-36% of all non-ischemic cardiomyopathies are attributed to alcohol. The prevalance of alcoholic cardiomyopathy in addiction ...

    • 2023/08/08
  4. Jun 1, 2015 · Background—Excessive alcohol consumption is associated with cardiomyopathy, but the influence of moderate alcohol use on cardiac structure and function is largely unknown. Methods and Results—We studied 4466 participants from visit 5 of the Atherosclerosis Risk in Communities (ARIC) study (76±5 years and 60% women) who underwent transthoracic echocardiography, excluding former drinkers ...

    • Alexandra Gonçalves, Pardeep S. Jhund, Brian Claggett, Amil M. Shah, Suma Konety, Kenneth Butler, Da...
    • 2015
  5. Dec 20, 2021 · There is a paucity of evidence investigating the relationship between long-term alcohol consumption and VA, and the studies that have been conducted are in select patient populations and have demonstrated inconsistent findings. 10–12 In a case-control study of ST-segment elevation myocardial infarction patients, consumption of >96 g of alcohol per week was associated with an increased risk ...

  6. Furthermore, potential beneficial effects of non-heavy alcohol consumption on CVD endpoints, as described in this review, have already been observed at very low levels, such as 100 g pure alcohol per week, which, at the lower end, translates to about 1 drink every other day. As such, most drinkers should drink less.

  7. Introduction. Long-term heavy alcohol consumption is known to have a direct toxic effect on the heart that can ultimately result in clinically diagnosed alcoholic cardiomyopathy.1 However, there will be a continuum of changes in cardiac structure and function in response to heavy drinking which may or may not ultimately result in a diagnosis of cardiomyopathy per se.

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