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  1. Oct 27, 2022 · This article reviews human research investigating neurobiological and psychological changes related to alcohol misuse that are associated with greater distress and stress-related alcohol craving and their role in predicting risk of binge drinking, relapse, and impact on treatment outcomes.

  2. May 24, 2021 · Stress leads to detrimental health outcomes through direct biological and indirect behavioural changes. Stress can lead to disruption to normal eating behaviours, although the strength of these associations is unknown.

    • Deborah C Hill, Mark Conner, Faye Clancy, Rachael H Moss, Sarah Wilding, Matt Bristow, Daryl B O'Con...
    • 2021
  3. Weight-related adaptions of the metabolic, neuroendocrine, and neuronal pathways can together potentiate food preference, craving and intake under conditions of stress. A sensitized feed-forward process may result in changes that promote elevated desires for and increased consumption of hyperpalatable foods.

  4. Oct 15, 2022 · Taken together, stress and stress system alterations by alcohol consumption could be associated with biased information processing, increased impulsivity and impaired control functions; a pattern that is known to be a key mechanism in the development of excessive alcohol use [46, 47].

  5. This study presents a novel application of the cognitive processing model of alcohol craving (CPMA) from the addictions field to the role of food craving in the well-established restrained eating–overeating relationship associated with binge eating disorder (BED).

  6. Higher present stress coping co-occurred with higher anticipated stress coping (r = .538, p 1 → 2 < .001, p 2 → 1 < .001) and with lower worry (r = −.156, p 1 → 2 < .001, p 2 → 1 < .001). Moreover, hunger co-occurred with food cravings (r = .384, p 1 → 2 < .001, p 2 → 1 < .001). No contemporaneous associations of goal-congruent ...

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  8. Oct 21, 2023 · Negative urgency often co-occurs with alcohol misuse and may be an endophenotype of AUD [106, 116]. Negative urgency is a risk factor for the initiation and exacerbation of alcohol use in young people during stress or depression, exemplified in studies of school- and college-age transitions [112, 115,116,117,118,119,122].

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